The Hawkseye 

May 2006 - page 8

 

Reflux continued...

Singers are particularly predisposed to reflux through sustained diaphragmatic compression of the stomach during “supported” singing, frequent late night eating, and performance stress.

Laryngopharyngeal reflux versus gastroesophageal reflux

Gastroesophageal reflux disease (GERD) is presently a more conspicuous media presence (e.g., “the Purple Pill”) than its relatively unknown cousin, laryngopharyngeal reflux. It is important to note that many patients suffering from LPR do not present with esophagus-related heartburn, or stomach upset/ulceration. Indeed, many patients are prompted to respond “I don’t have reflux because I don’t have heartburn!” when first diagnosed with LPR. Reflux may selectively affect relatively delicate throat tissues to the exclusion of the tougher esophagus. Although GERD and LPR may be considered as separate diagnostic entities by many treatment specialists, medical recommendations for both kinds of reflux are similar. There are no statistics on the related incidence of GERD to LPR, and one condition is not necessarily a precursor to the other.

Causes of laryngopharyngeal reflux

The upper esophageal sphincter is a muscular valve that primarily facilitates food entry into the gullet. The lower esophageal sphincter primarily contains the digestive process within the stomach. Secretions must pass through both of these valves to cause LPR; mechanically through factors such as obesity-related abdominal pressure, inverted posture, and carbonated beverages. Smoking and certain foods may relax the lower esophageal sphincter, and spicy or acidic foods may compound existing tissue irritation.

Because of the muscular esophagus involvement, it is possible that inherent weakness or transient muscular relaxation may allow reflux seepage. In a contrasting manner, increasing one’s stress may overly activate the digestive system and precipitate reflux.

Late eating, within three hours of bed, has been significantly implicated in reflux activity. Hiatal herniation, in which the upper stomach protrudes through the diaphragm, has been implicated in reflux activity, but neither showing to be the cause of the other has yet been established.

 

Research into the linkage of the digestive and respiratory systems has yielded solid evidence of relationships between reflux and asthma, and even more remotely, between reflux and inner ear inflammation. These far reaching reflux effects are logical when one considers that the stomach is an intensely harsh environment in which from three to four and a half liters of hydrochloric acid are secreted on a daily basis . . . in addition to digestive enzymes. Somewhat surprisingly, an acidic environment is not necessary for the effective digestion of food; note that modern reflux medications are very effective at suppressing acid production. The purpose of stomach acidity is to comb out potentially harmful microbes in our food, and activate pepsinogen into the primary digestive enzyme, pepsin.

Treatment of laryngopharyngeal reflux

A proton pump inhibitor (PPI) suppresses the chemical messenger that stimulates acid production factories (proton pumps) within parietal stomach cells. This powerful class of medications used include: Nexium, Prilosec/Omeprazole, Prevacid, AcipHex, and Protonix. PPIs produce the most adverse reactions in users; 1% experience headache, diarrhea or constipation, abdominal pain, flatulence (gas), nausea/vomiting, or dry mouth.

Relatively less aggressive and effective histamine blockers such as Zantac, Tagamet, and Pepcid exert a different mechanism of acid control via the jamming of intra-cellular proton pumps. Both PPIs and histamine blockers are effective for between ½ and ¾ of a 24 hour period, therefore double dosing each day may be necessary.

Common antacids buffer secreted acid, and are the least aggressive medications for reflux intervention. Antacids may also create a foam barrier in the lower esophagus and upper stomach which aids in reflux containment for up to ninety minutes. Basic recommendations specify head of bed elevation and antacid use prior to sleep.

There may be additional merit in a drug such as Pepcid Complete which combines both a histamine blocker and antacid. Likewise, it is possible to combine a PPI and histamine blocker (e.g., Nexium in the morning and Zantac at night), or double dose each class.

All the aforementioned medication classes are now available over-the-counter; however, it would be most 

Reflux continued on page 9


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